Michael Rauchman, M.D.
Department of Internal Medicine-Nephrology
Understanding the molecular genetic basis of mammalian kidney development and how disruption of specific pathways leads to abnormal development of this organ.
Office: DRC, Room 405
Voice: (314) 977-9366
M.D.C.M., 1984, McGill University Faculty of Medicine
Our lab focuses on understanding the molecular genetic basis of mammalian kidney development and on how disruption of specific pathways leads to abnormal development of this vital organ. We use the mouse as our model system.
- Conditional expression of wnt9b in six2-positive cells disrupts stomach and kidney function.
Kiefer SM, Robbins L, Rauchman M. PLoS One. (2012) 7(8):e43098.
- Tamm-Horsfall protein-deficient thick ascending limbs promote injury to neighboring S3 segments in an MIP-2-dependent mechanism.
El-Achkar TM, McCracken R, Rauchman M, Heitmeier MR, Al-Aly Z, Dagher PC, Wu XR. Am J Physiol Renal Physiol. (2011) 300(4):F999-1007.
- Early nephrologist involvement in hospital-acquired acute kidney injury: a pilot study.
Balasubramanian G, Al-Aly Z, Moiz A, Rauchman M, Zhang Z, Gopalakrishnan R, Balasubramanian S, El-Achkar TM. Am J Kidney Dis. (2011) 57(2):228-34.