Michael Rauchman, M.D.C.M.
Department of Internal Medicine-Nephrology
Understanding the molecular genetic basis of mammalian kidney development and how disruption of specific pathways leads to abnormal development of this organ.
Office: DRC, Room 405
Phone: (314) 977-9366
M.D.C.M., 1984, McGill University Faculty of Medicine
Our lab focuses on understanding the molecular genetic basis of mammalian kidney development and on how disruption of specific pathways leads to abnormal development of this vital organ. We use the mouse as our model system.
- Notch-Tnf signaling is required for development and homeostasis of arterial valves.
Wang y, Wu B, et al. Eur Heart J. (2015) Oct 21 [Epub ahead of print].
- A mouse model of Townes-Brocks syndrome expressing a truncated mutant Sall1 protein is protected from acute kidney injury.
Hursch S, El-Achkar T, et al. Am J Physiol Renal Physiol. (2015) 309(10):F852-863.
- Acute kidney injury after cardiac surgery: Is minocycline protective?.
Golestaneh L, Lindsey K, et al. J Nephrol. (2015) 28(2):193-199.
- The nucleosome remodeling and deacetylase complex in development and disease.
Basta J and Rauchman M. Transl Res. (2015) 165(1):36-47.